Vaccine Effects On SPIFR Programs  

Reasons why COVID vaccines may not work

There are a number of peer-reviewed publications now providing reasons why mRNA vaccination could potentially cause an increased risk of COVID infection, or have other risks:

1. The well reported drop in white cell count (neutrophyls) immediately following vaccination.

2. Immune focus on a single antigen (spike protein) and associated variant selection causes vaccine break-through. The explosion in the number of new variants following vaccination is being monitored by the international collaboration of scientists, Nextstrain. There are millions of complete SARS-CoV-2 genomes available and this number increases every day.

3. Antibody dependent enhancement; anti-SARS-CoV-2 antibodies could actually exacerbate COVID-19 through antibody-dependent enhancement (ADE). ADE is a reminder that not all antibody responses are helpful and some can be harmful.

4. Vaccine induced auto-immune responses including VITT. As reported by JAMA Neurology, circulating platelet-activating platelet factor 4 (PF4) antibodies have been found in many patients suffering from vaccine-induced immune thrombotic thrombocytopenia (VITT). This is probably caused by injection components binding with the person's own platelet factor to create a 'neo-antigen'. This neo-antigen is what then causes an immune response resulting in clotting, intracerebral haemorrhage and the death of around half of those people affected.

5. Natural immunity provides better protection; there is increasingly overwhelming evidence that natural immunity is more powerful and broader in terms of preventing infection. The largest study yet comparing COVID vaccines to natural infection has produced results that would have surprised no one 50 years ago.

6. A jab in the arm can't work. According to the Journal of Immunology, "The route of vaccination is important in influencing immune responses at the initial site of pathogen invasion where protection is most effective". Molecular immunologist, Professor Edward Steele points out that a jab in the arm cannot protect against a mucosal viral invasion - purely on the basis of first principles. It is an antibody known as IgA predominantly found in our nasal, lung and gut mucosa that is responsible for the first line of defence against respiratory infections. Unless the IgA is activated the vaccine simply can't work. This observation would seem to demonstrate a fairly fundamental flaw in the inoculation method.

7. Vitamin D deficiency; T-cells can only fight viral infections when there is sufficient vitamin D present. When a T-cell is exposed to a pathogen such as a virus, it extends a signalling device or 'antenna' known as a vitamin D receptor, with which it searches for vitamin D

8. The spike protein may itself toxic and prevent DNA repair; a recent study that looked at what actually happened to the spike protein has demonstrated a potential molecular mechanism by which the spike protein might impede adaptive immunity and underscore the potential side effects of full-length spike-based vaccines. The spike protein appears to localise in the nucleus, and here it inhibits DNA damage repair by impeding key processes.

9. According to the Journal of Immunology Original Antigenic Sin, or OAS.